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The age old tension remains taught


Nothing about this makes any sense. We’ve already got a number of people pointing out flaws like why did he wait 15 years to write about it, why does it look like it was written by an LLM, and is it really reasonable to blame such a massive failure completely on your peers and not take an ounce of responsibility yourself? But these things all start to make sense once you actually reach the end of the article and realize it’s all a ploy to sell you his fancy new equivalent to a self-help book, which you can tell is legit because its name is a forced acronym. Can we take this off the front page please?


I think it is better to be charitable. I think he does genuinely believe what he wrote is what happened. His PDF book is free and Creative Commons.

There could be many reasons he waited this long. Maybe he waited until he was retired and would not face blowback. Maybe he just has some free time.

It is very plausible that WebOS could have been an equal peer to iOS and Android. CEOs have killed off projects that might have been great commercial successes while perusing short term gains.

In a decade's time we might hear a story from inside ATI or AMD how they killed off their chance of beating CUDA for short term gains.


> Can we take this off the front page please?

Don’t do this. Engagement is what drives stories to the front page. If you don’t like it just move on.


It must be X given that they recommend installing xbacklight, arandr, and Compton alongside it.


And the section on running in Xephyr (X server that runs inside another X session) for debugging.


Was this written by an LLM?


If you mean the comment, I think so. That's why it is probably flagged


No, it was not written by a language model (LLM). I know one of the authors, Mingzheng Wu, and have followed his work closely, including his papers on the rapid enhancement of glutamate-evoked dendritic spinogenesis in the medial prefrontal cortex through dopaminergic mechanisms, and the suppression of neuropathic pain through inhibition of YAP/TAZ activity in the spinal cord.


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